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Nandrolone (Deca Durabolin) Nandrolone is one of the most commonly used steroids for muscle growth. Nandrolone exerts its actions via its binding to CB1 receptors. In the present study, we used a stereoselective binding assay, utilizing the synthetic cannabinoid-1A receptor (CB1A) and the human CB1 receptor antagonist [ 3 H]WIN55212, nandrolone cz. Nandrolone-induced ERK phosphorylation was significantly reduced in CB1A mice, but not in CB1A−/− mice. Our results suggest that nandrolone exerts its steroidogenic activity via CB1 receptor-mediated ERK phosphorylation-independent action, steroids for muscle aches. The potential of nandrolone to stimulate muscle growth via its antiestrogenic action on estrogen receptor (ER) is well established (Liu et al., 1991; Gao et al., 2007). Indeed, it has been shown that nandrolone is a potent and selective ER antagonist, with very weak agonist actions in ERα-containing cells (Lu et al., 2003). However, the specific effects of nandrolone on ERα/β are unknown, nandrolone cz. In the present study, we examined the effects of nandrolone administration on ERβ/ERα maturation in HEK293T cells, anadrol oral steroid. We assessed the effects of nandrolone on the maturation of ERβ/ERα by using the E1-MEM method (Santosh et al., 2011) which used the fluorescent antibody 4′,6-diamido[3a,4b]pyridinium bromide (Sigma-Aldrich). Although the specificity of 4′,6-diamido[3a,4b]pyridinium bromide (Sigma-Aldrich) was well-known previously (Hudson et al, anabolic steroids schedule., 2006), nandrolone administered to rats in the presence of ERβ/ERα or ERβ/ERα/ERα ERK ligands (saline, prazosin and prazosin-6-chloro-5-chloro-1-hydroxy-α-methyl-3-methoxyphenyl) in a dose-dependent way, did not affect the maturation of ERβ/ERα ( ), anabolic steroids schedule. Furthermore, neither nandrolone-precursor nor nandrolone-preset compounds affected the maturation of ERα/ERβ.
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